Ungleichgewicht der Eierstock-Nebennieren-Schilddrüsen-Achse (ENS-Achse)

Abbreviated version

The hormonal system consists of eight different glands, which are strategically distributed in the body. Three of the most important for women are the ovaries, adrenals and thyroid gland.

The control of the adrenal and ovarian systems begins at the hypothalamus. The adrenal glands in particular are regulated via the hypothalamic-pituitary-adrenal axis (HHN axis). The ovarian hormones are regulated via the hypothalamic-pituitary-ovarian axis. Each of the end organs (adrenals, thyroid and ovaries) produces several hormones that continue to exert their influence on the rest of the body. In addition, each of the end organs is hormonally involved in other organ systems via a number of networks or axes.

One of these complex hormonal axes links the adrenals, thyroid and ovaries. This axis is called ovarian adrenal gland axis (ENS axis, English: Ovarian adrenal thyroid axis or OAT axis). What happens in one organ will affect the other organs physiologically, clinically, or subclinically. These three organs are therefore hormonally closely interdependent in order to function optimally.If the adrenal glands are weak, then the thyroid is often disturbed in function and the menstrual cycle is irregular. Similarly, hypothyroidism often makes adrenal fatigue worse. Finally, those who suffer from hormonal imbalance of the ovaries, such as estrogen dominance, often exacerbate existing subclinical hypothyroidism.

All three organs of this axis must be in optimal balance for a woman to feel comfortable. It’s like a three-legged chair, with all three feet in perfect balance so you can sit on it safely. An imbalance in the ENS axis leads to a myriad of constitutions that are troublesome in their mild form and that lead to disability in their severe form.

OAT Axis

The common complaints of those who suffer from an imbalance of the ENS axis include a mixture of symptoms suggestive of concomitant adrenal, thyroid and ovarian decompensation. , The symptoms present often, but not always, indicate adrenal insufficiency, estrogen dominance or hypothyroidism. These may commonly include: sleep disorder, fatigue, chronic fatigue, joint pain, exercise intolerance, drowsiness, sugar intolerance, diabetes, dry skin, feeling cold, slow metabolism, inability to lose weight, PMS, endometriosis, irregular menstrual cycle, fibrocystic mastopathy, anxiety, depression and accumulation of Fat around the waist.

Those who have had their ovaries removed may still have an ENS axis imbalance because of estrogen imbalances and ovaries are not the only sites where estrogen is produced. The adrenal glands as well as the adipose tissue (fat cells) are also sites where estrogen is actively produced.

An ENS-axis imbalance represents a state of hormonal axis irregularity that still needs to be recognized by conventional medicine. It may be subclinical or clinical. Because its clinical presentation is an interplay of multiple imbalanced hormonal systems, there is no definitive test that can isolate and pinpoint this imbalance condition. A formal and extensive research is needed. Our current understanding of this condition stems from clinical experience and case studies. As such, an ENS-axis imbalance should currently be more appropriately considered a clinical condition rather than a disease condition. This multi-organ clinical condition, the frequent imbalances of the ovarian,

Since the myriad of symptoms are entangled and diverse, physicians, even those who are oriented toward naturopathic medicine, can easily be overwhelmed.

In males, this imbalance condition is also associated predominantly with low libido and testosterone replaces ovarian imbalance. Low androgen levels also affect the thyroid and adrenal functions.

Estrogen rule and ENS axis

Estrogen dominance and ENS-axis estrogen dominance can increase thyroid binding proteins in the bloodstream. Thyroid blood test results may therefore be normal, although too few thyroid hormones are present in the tissue, resulting in a condition of subclinical or clinical hypothyroidism.

When estrogen levels are high , the adrenal cortex can no longer respond to signals from the brain. In other words, although the brain requires the production of more cortisol, the adrenal glands respond poorly to the request. The result is that cortisol production is suboptimal compared to the request signal. In addition, estrogen interferes with adrenal function by disrupting the secretion of cortisol by the adrenal cortex. High levels of estrogen may cause a corresponding increase in cortisol-binding globulin levels. The cortisol-binding globulin in turn impairs hormone function and circulates in the bloodstream, where it binds to the cortisol, rendering it inactive. That’s whyFor example, a woman with estrogen dominance may have adequate total cortisol levels in the bloodstream. Blood tests to examine their total cortisol levels may indicate a value that is within the normal range, but their level of available cortisol may be too low. Since only free cortisol can pass through the cell membranes and activate receptors within the cells, the effectiveness of cortisol is weakened at the cellular level.

Just as estrogen dominance can contribute to adrenocortical insufficiency, adrenocortical insufficiency can contribute to estrogen dominance. Cortisol is formed in the adrenal cortex from progesterone. If the adrenal glands are too weak, there is a tendency to lower the release of progesterone in favor of cortisol. The result is often a low level of progesterone, leading to a condition with relative estrogen dominance, with the many undesirable consequences mentioned above. This creates an unfavorable feedback loop and a vicious circle. Too much estrogen affects both thyroid and adrenal function. Thyroid dysfunction and adrenal fatigue in turn make estrogen dominance even worse.

Adrenals and ENS axis imbalance

The adrenal glands are usually the first of the endocrine functions that fail when stress has overwhelmed the body’s normal compensatory response. Unfortunately, this is rarely recognized as a pathological condition. Accepted societal compensatory actions such as coffee intake often mask the subliminal problem as the adrenal glands are over-regulated to mask the first signs and symptoms of adrenal fatigue. The next endocrine gland that is affected is the part of the pancreas that produces insulin. The blood sugar of the body gets into an imbalance and this malfunction is temporarily corrected by the intake of sweet drinks, energy drinks and donuts. After the pancreas, the thyroid gland comes. The fact that they feel lethargic, cold and growing is the predominant symptom that causes patients to see their doctor, usually the first time. This is the time when thyroid hypofunction is first detected. Thyroid medications are routinely prescribed. Nonetheless, more than 70 percent of patients taking thyroid medications continue to experience symptoms even after a long time. Symptoms of estrogen dominance occur together with hypothyroidism, a condition that indicates a malfunction of the ovarian system. Symptoms include PMS, endometriosis, lumps in the chest, and an irregular menstrual cycle. Hormone replacement medication can be successful in the short term, but if the adrenal glands are not treated first, patients will often respond poorly and eventually stop responding. Finally, the parathyroid glands, the pineal gland, the autonomic nervous system and the hypothalamus are affected. At this time, the ENS axis is severely affected.

Adrenal insufficiency is often the last to be discovered and only seriously considered when the patient is already seriously decompensated, with serious involvement of the ovaries and the thyroid. Even if discovered, too much attention is paid to the often coexistent ovarian and thyroid imbalances, which is now a full-blown ovarian-adrenal-thyroid-axis imbalance.

The key to this imbalance is in the adrenal glands, where cortisol is regulated. In adrenal insufficiency, an internal cortisol imbalance often results in multiple organ failure, including thyroid and ovaries. Adrenal insufficiency is an important common and often overlooked cause of secondary clinical or subclinical hypothyroidism. Usually the patient comes to the doctor’s office and complains of tiredness, dry skin, weight gain, low body temperature and a sleep disorder. Thyroid laboratory test results show either a normal or high TSH value, a normal or low value of T3 and free T3, a normal or low value of T4 and free T4. The patient is usually prescribed various thyroid medications. The patient may feel a temporary improvement, but in the end the improvement will fail as there is little recovery and the symptoms persist. An excess of thyroid medicine may be prescribed to normalize what appears to be suboptimal blood levels. There is an overload of the body, as the basic metabolism rate is increased. This can mask existing adrenal fatigue, exacerbate existing adrenal fatigue, and trigger adrenal fatigue and adrenal crisis. Antidepressants are often prescribed as a solution to control the symptoms, as the doctors have ideas about how to help the sufferer. This rarely works,

It is important to realize that optimal adrenal function plays a key role in this imbalance. When the adrenal glands are weak, causing cortisol, then the organ resistance affects almost all organs that are hormone-regulated, including the ovaries, the thyroid, and the pancreas. Few hormones can work at their optimum level in the case of adrenal fatigue. A variety of hormones, including insulin, progesterone, estrogen and testosterone, are compromised.The normal negative feedback loop may be weakened and the binding of carrier hormones to the blood may be disturbed. The ability of each hormone to regulate and fine-tune its target organ in order to achieve homeostasis is therefore often compromised. Blood pressure can become uneven, blood sugar levels can fluctuate greatly, bipolar states and anxiety come and go, reactive adrenaline gets uncontrollable, drowsiness increases, metabolic functions slow down, and menstrual flow becomes irregular.

Because of their lack of adrenal fatigue training, doctors often treat the symptoms of weak thyroid and ovarian systems and pay no attention to adrenal gland dysfunction. Let’s see how that can lead to devastating effects. In the early stages of adrenal insufficiency, cortisol release is high as the body tries to neutralize stress by producing more of it. However, producing too much cortisol will have several undesirable effects. For example, cortisol blocks the progesterone receptors so they are less responsive to progesterone. The progesterone production, which is normally done by the adrenals, is stopped and instead cortisol is produced.

Inadequate progesterone production leads to an imbalance between estrogen and progesterone. With too little progesterone to balance the estrogen, the body can experience estrogen dominance. It is no coincidence that we experience a proliferation of constitutions associated with too much estrogen, such as PMS, fibroids and premenopausal syndromes, when women are in their mid-thirties and early-forties.

Many chronically ill patients have both weak adrenal and thyroid functions. In such cases, it is important to begin the recovery process by supporting the adrenal glands before increasing the thyroid hormone. Otherwise, increased thyroid hormone levels could put even more strain on the already weak adrenal glands, leading to adrenal collapse and even more decompensation. Recovery plans that focus on the malfunction of a single organ without considering the axis imbalance often fail and can even worsen the condition.

Attempts to rebalance the ovarian and thyroid hormones without paying close attention to the adrenal hormones often fail. Conversely , an initial focus on the adrenal glands often leads to spectacular results, as the ovarian hormones and thyroid hormones naturally regain their balance when the adrenal glands recover.

Hypothyroidism

The thyroid gland acts like the body’s barometer. Its main function is to help cells convert oxygen and calories into energy. It regulates heart rate, blood pressure, body temperature, metabolism, and growth.

More than 10 million Americans have been diagnosed with thyroid disease and another 13 million people are estimated to have undiagnosed thyroid problems. About 10 percent of the adult population is afflicted with this frequently overlooked disease of epidemic proportion. A dysfunctional thyroid can affect almost every aspect of health. It is one of the most under-diagnosed hormonal imbalances of aging, along with estrogen dominance, and adrenal fatigue. It is estimated by age 50, one out of every twelve women wants to have some degree of hypothyroidism. By age 60, it’s one out of six. In fact, among the elderly, hypothyroidism is sometimes misdiagnosed as dementia.

The thyroid gland, under TSH signal, secretes two essential thyroid hormones: triiodothyronine (T3) and thyroxine (T4), which is responsible for regulating cell metabolism in every cell in your body. A healthy person secretes all the circulating T4 (about 90 to 100 mcg daily) and about 20 percent of the circulating T3. The T4 made by the thyroid gland circulates throughout the body and is converted into approximately equal amounts of T3 and reverse T3 (rT3). Most of the biological activity of thyroid hormones is due to T3. It has a higher affinity for thyroid receptors and is about four times more potent than T4. RT3 acts as a braking system to T3.Not only is it inactive (having only 1 percent of T3 activity), it binds to T3 receptors and blocks the action of T3. T4 should therefore be considered a pro-hormone and precursor to T3 and rT3. Normal physiological production ratio of T4 to T3 is 3.3: 1. A proper thyroid gland requires a perfect balance of T4, T3, and rT3. The T3 / rT3 ratio is one of the most useful markers for true hypothyroidism and diminished cellular function.

Anti-thyroid effects, over-consumption of uncooked “goitrogenic” foods, such as broccoli, turnips, radish, cauliflower and brussels sprouts, adrenal insufficiency or fatigue, mercury intoxication (amalgam are 50 percent mercury), auto-immune diseases and infection.

Hypothyroidism may be primary or secondary. Primary hypothyroidism can be treated by administering thyroid medications. If thyroid hypofunction symptoms such as low body temperature, fatigue, dry skin, and weight gain persist despite taking thyroid hormones, then whatever the lab values, the cause of low thyroid needs to be sought elsewhere.

Secondary hypothyroidism is caused by the malfunction of another organ system. One of the often overlooked causes is adrenal fatigue. Adrenal insufficiency is perhaps the most common cause of secondary hypothyroidism, both clinical and subclinical. Weak adrenal function often results in weak thyroid function, usually characterized by a high level of thyroxine-binding globulin (TBG), low free T4, low free T3, high TSH, and low body temperature. Few doctors are trained to make this connection. Fortunately, secondary hypothyroidism can be reversed if the underlying problem (such as adrenal insufficiency) is resolved.

Thyroid and ENS axis thyroid imbalance

Thyroid and ENS-axis thyroid imbalance is perhaps the most confusing, complex and difficult-to-treat endocrine dysfunction. Attempts to treat thyroid symptoms without fully understanding the actual complex cause behind them, with the connection to the ovarian and adrenal system, therefore often fail over time.

The thyroid is closely linked to the ovarian and adrenal system. By regulating the metabolism, it affects the reproductive gland activity. The thyroid affects the production of SHBG (sex hormone binding globulin), prolactin and GnRH (gonadotrophin-releasing hormone), all of which affects the menstrual cycle and the ability to become pregnant. The thyroid hormones also stimulate the production of progesterone in the ovaries.

Thyroid disorders can lead to disruption of the menstrual cycle in younger women as well as in more mature women.Women with PCOS and infertility problems often have chronically too little progesterone. In premenstrual women, these problems are often aggravated. Untreated thyroid problems could contribute to infertility, PMS and menopausal symptoms. In addition, thyroid hormones are similar to certain metabolites of estrogen and progesterone, and receptors for the uptake of thyroid hormones may be blocked or the uptake may be facilitated by estrogen and progesterone. Imbalances of the thyroid hormones T3 and T4, combined with estrogen and progesterone imbalances, can mimic the symptoms of menopause, resulting in various effects in the areas of mood, temperature regulation, fluid retention, energy and sleep.Women with normal TSH levels may actually be in a state of subclinical hypothyroidism without knowing it.

The thyroid gland is also closely linked to the adrenal system. When the adrenals are exhausted, the ability of the adrenal glands to deal with the stress associated with the body’s normal functions and energy requirements is often compromised. To improve survivability, the adrenal glands regulate energy production. In other words, the body’s metabolism is down-regulated to save energy. The body has to rest. The body therefore has several options to reduce energy production and to improve survival under the guidance of the adrenal glands.

The laboratory test results for T4 and T3 may be normal, and the classic symptoms of hypothyroidism are manifest with a persistent low body temperature and a slow ankle reflex. On the other hand, the laboratory test results for free T4 and free T3 may be low while the TSH value is normal or high. In both scenarios, thyroid hormone replacement treatment with T4 and T3 without first considering adrenal reinforcement is a common mistake and often causes the ENS axis imbalance to worsen.The reason is simple. Thyroid hormone replacement treatment tends to increase the metabolism. Increasing the basal metabolism is similar to the simultaneous oversteer of all systems of the body when the body tries to rest by adjusting down the many mechanisms previously described. The survival mechanism of the body is designed to achieve a reduction rather than an increase in T4 and T3 levels. What the body wants (shut down) and what the medicines are intended for (start up) are diametrically opposed.

To administer thyroid medications in case of advanced adrenal insufficiency without at the same time paying attention to an adrenal recovery often fails. In many cases it is similar to pouring oil on fire. An already weak adrenal system in a low energy state may not be able to meet this additional energy requirement. What the adrenals need is rest, no extra work.

Thyroid medicines administered under such conditions may initially lead to a temporary relief of symptoms and a slight boost in energy. This is often short lived. Finally, the tiredness comes back, as the thyroid drug additionally exacerbates the existing adrenal fatigue and often causes an adrenal crisis. The general fatigue is getting worse, far beyond what the medication is trying to combat. Only by increasing the drug dose or by switching to a stronger thyroid medication can it be prevented that the fatigue worsens.

Let’s take a closer look. While the laboratory values ??of T4, T3 and TSH act as if they have improved since the thyroid medications were administered, the patient does not show any clinical improvement and his condition often worsens over time. Doctors may be led astray by the lab results “improving”, meaning that they are “on the right track” and not be prepared for the fact that a possible concomitant adrenal malfunction is the main culprit behind it. If the FT4, FT3, and rT3 values ??are not considered in the clinical picture, the true cellular delivery of thyroid medication is unknown.The cry for action of the body, by enforcing a permanent low body temperature, is not complied with. The unsuspecting physician may increase the thyroid drug dose more and more in an attempt to relieve the unpleasant and unresolved thyroid hypofunction symptoms. This approach rarely works long-term, as previously mentioned, and unknowingly exposes the patient to unintentional negative consequences on the adrenal glands, overshadowing any benefit to the thyroid gland. As adrenal glands continue to function, they will continue to down-regulate as much as possible, which will weaken the body’s response to thyroid medication.Over time, despite improved or stabilized T4, T3, and TSH levels that might be considered normal, the patient needs an even larger clinical dose of the drug to suppress the symptoms. The patient continues to struggle with unresolved symptoms and low body temperatures that refuse to normalize, while the classic signs of hypothyroidism persist despite medication.

So it’s no surprise that 70 percent of patients taking thyroid hormones continue to complain about the symptoms of hypothyroidism. Finally, the body of the patients is overcharged with thyroid medication, while unfavorable side effects such as palpitations and tremors come to light and the patient continues to feel tired and sluggish. This condition can be described as “tired and still unable to sleep”.

Those who have hypothyroidism, but whose condition does not improve after taking thyroid hormones, should therefore always investigate whether adrenal insufficiency is a potential aetiology for their thyroid problem. The normalization of adrenal function is the key in such cases and often leads to a spontaneous resolution of hypothyroidism symptoms. The faster the patient recovers from adrenal fatigue, the faster the symptoms of hypothyroidism are resolved.

It is important to note that laboratory tests of thyroid function will continue to display low thyroid levels for some time during this adrenal-focused thyroid recovery due to a lagging effect. The TSH value could still remain high and out of the normal range, while the free T3 and free T4 could remain low. This lagging effect can go on for months. However, as the adrenals recover, the patient will experience clinical improvement, with rising body temperature to normal levels, more energy, less need for thyroid medication, and improved weight management. Conventional doctors who are not trained to recognize this adrenal-thyroid connection are clinically pleasantly surprised to see their patients recover, despite the abnormal lab results, but without knowing why. The key to this, of course, lies in the improved adrenal function.

While it is important to focus primarily on supporting the adrenal glands in those with concomitant adrenal and thyroid dysfunction, it is important not to abruptly stop any thyroid medication (and other natural supplements that may have stimulant effects, such as Herbs or glandular preparations) without professional guidance. An abrupt adjustment can lead to unpleasant and unbearable withdrawal symptoms. In rare cases, an adrenal crisis can be triggered. The best clinical strategy is to focus on adrenal care and restore thyroid function by itself.

The flip side of the coin is that weak adrenal glands and a weak thyroid gland, if not treated simultaneously, form an unfavorable, augmented downslope spiral. Those who need an ever-increasing dose of thyroid medication to combat fatigue may end up being dependent on strong thyroid medications, but suffer from the side-effects of thyroid toxicity (such as palpitations) as the dose increases. You feel tired all the time, but you still can not sleep, as already mentioned, and you feel anxious all day long. Inside, the adrenal glands become weaker and weaker, just as the stimulant properties of the thyroid drugs are negated by the constant and overwhelming rejection of the adrenal glands, which causes the body to become more and more down-regulated to save energy. This is the worst possible situation. It happens too often and is not noticed.Both physicians and patients are perplexed by what looks like clinical inconsistencies in which the TSH laboratory values ??are improved (as a result of increased medication) or high TSH levels refuse to go down, which increases the need for thyroid medication for energy Adrenal insufficiency worsens, with continued low body temperature, metabolic imbalances, weight gain, and increasing inertia.

Finally, it is important to note that regular weight loss protocols often fail when there is an ENS-axis imbalance due to an underlying metabolic disorder. The good news is that when the axle imbalance is resolved, it often results in natural weight loss. Therefore, making weight loss one of the most important recovery priorities is usually a strategy that fails.

Hypothyroidism, adrenal fatigue and ENS-axis imbalance

Of those taking thyroid hormones, 70 percent still complain of symptoms. Often, physicians do not take the time they need to understand how adrenal fatigue can be one of the major causes of secondary hypothyroidism, either clinically or subclinically. That one relies too much on laboratory test values ??as a yardstick for thyroid hormone replacement is very common. Thyroid function tests may indicate values ??in a normal range, but the condition of the patient may not improve. The doctors are misguided by thinking that a higher dose of medication might be needed.In a situation with concomitant adrenal fatigue and hypothyroidism, this is often a strategy that fails. Higher thyroid medications are only used to mask the underlying adrenal insufficiency. Over time, the patient gets worse and worse despite an ever-increasing dose of medication. As the thyroid drug dose increases, the patient may experience palpitations and anxiety. At the same time, however, reducing the drug dose is difficult due to the potential withdrawal problems.

It is not uncommon to have symptoms of low adrenal gland and low thyroid function at the same time. In fact, that’s the norm and not the exception. Traditional medicine often does not see it because of ignorance about adrenal fatigue. Those who are diagnosed with hypothyroidism after a traumatic and stressful event such as pregnancy, accident, infection or an emotional trauma such as divorce or death of a loved one should be especially vigilant if thyroid hormones alone do not help.

Those with poor body temperature regulation tend to be candidates for a mixed representation. Having the symptoms of adrenal insufficiency as well as hypothyroidism should alert us that both adrenal fatigue and hypothyroidism may be present at the same time. This is especially common among those with an ENS axis imbalance.These individuals may show a persistent low body temperature from 32 ° C to just below 37 ° C. They may also show a slightly exaggerated response of body temperature compared to ambient temperature, which is characterized by a heat sensation when it is warm and a cold sensation when it is cool. They also suffer from fatigue, dry skin and weight loss.

Those who have mixed type representations offer the biggest challenge. Solving the adrenal component should take precedence and is the key to a complete healing process. The normalization of adrenal function in such cases will often lead to a spontaneous and dramatic resolution of thyroid hypofunction symptoms.

Axis component dominance

In an ENS axis imbalance, it often happens that one of the components of the axis is clinically more dominant and therefore more problematic. The imbalance between the thyroid, adrenals and ovaries is usually not the same. The clinically dominant part usually reflects the organ system, which is the weakest in terms of constitution and thus the most affected. For example, the symptoms of subclinical hypothyroidism may be more severe as compared to adrenal and ovarian dysfunction.

Clinically , those who are thyroid-dominant usually have a seriously low level of physical energy in addition to the classic signs of hypothyroidism such as dry skin and the inability to lose weight. Your main clinical complaint is fatigue. For example, they are “too tired” to worry about PMS or to be depressed, even if the symptoms of depression are present. That’s a hint for health care providers. UnfortunatelyMost healthcare professionals do not see this unless they are trained for adrenal fatigue. Doctors are easily misled into thinking that the only problem is the thyroid gland without taking the others into account, resulting in incomplete diagnosis and ineffective therapy.

The adrenal-dominant type usually presents with emotional lability states such as anxiety and irritability. These individuals are also tired, but the fatigue is negligible compared to the rollercoaster ride they experience due to the anger and anger that are easily elicited. Those who are ovarian dominant present with marked drowsiness and memory loss in addition to PMS and other estrogen dominance symptoms.

Recognizing which of the ENS Axis components is dominant plays an important clinical role in designing a comprehensive recovery program, as nutritional support, dietary and lifestyle changes are different for each. The natural substance gaba is usually a good sleep aid for those who are adrenal dominant. However, it is not as effective when compared to 5-HTP for those who are thyroid-dominant. There is a special way for each nutrient to be optimally used, and it must fit the right condition for maximum effectiveness.

In terms of nutrition, the thyroid dominant type mostly benefits from a vegetarian diet with a high fiber content to increase gastric assimilation, while the ovarian type usually performs better with a diet high in protein compared to carbohydrates Share copes. The adrenal-dominant type is best served with a balanced diet with a tendency towards protein and fat, along with a higher meal frequency to cope with hypoglycaemia. Those of a mixed type need a combination of the above.

As far as movement is concerned, the thyroid dominant type works best with rhythmic exercises such as running and swimming. The ovarian dominant type works best with more gentle and mentally-focused exercises like yoga.The adrenal-dominant type is more complicated, as these individuals can be divided into two main categories, those with a strong adrenal function and those with a weak adrenal function. Energetic exercise is often helpful in the type with high adrenal function to reduce the excess of adrenaline. Those with a weak adrenal function should often refrain from any exercise until the body has regained its footing by supporting adequate nutrition. When the physical reserve has been increased, gradual strengthening of isometric and isotonic exercises may be considered.

Often, during the recovery process, the dominance type may also change. One can, for example, be thyroid-dominant and go in the direction of an adrenal dominance. This can happen if the thyroid function improves or if an acute adrenal breakdown overwhelms the thyroid gland. Knowing which component is dominant during the recovery period allows the clinician to prioritize the recovery plan and use appropriate natural support at the right time. Common self-directed or standardized shotgun schemes without taking into account the types of dominance and their current progress often results in a delay or failure in recovery.

Translated with the full permission of dr. Michael Lam (the “author”) by the Swedish Journal of Nutritional and Functional Medicine. The sole responsibility for the accuracy of the translation lies with the translator and the author assumes no responsibility for the accuracy of the translation. The original article in English can be viewed at www.DrLam.com